The effect of alcohol on blood glucose in Type 1 diabetes - Metabolic modelling and integration in a decision support system

Authors: Plougmann, S., Hejlesen, O., Turner, B., Kerr, D. and Cavan, D.

Journal: International Journal of Medical Informatics

Volume: 70

Issue: 2-3

Pages: 337-344

ISSN: 1386-5056

DOI: 10.1016/S1386-5056(03)00038-8

Abstract:

Introduction: We have recently shown, in studies with patients with Type 1 (insulin dependent) diabetes, that alcohol intake at 21:00 h significantly reduced blood glucose values after 10-12 h, compared with control studies with no alcohol. Hypothesis: We hypothesised that this was due to the following effects of alcohol: (1) alcohol metabolism increases NADH, leading to a reduction in hepatic gluconeogenesis; (2) increased glycogen phosphorylase activity depletes hepatic glycogen stores; (3) after the alcohol is metabolised, hepatic insulin sensitivity is increased, leading to the restoration of glycogen stores and reduction in blood glucose levels; and (4) consequently, after several hours, glycogen stores and insulin sensitivity return to normal. Results: A model describing these changes (DiasNet-Alcohol) was implemented into the DiasNet model of human glucose metabolism. Our study suggests that the DiasNet-Alcohol model gives a reasonable approximation of these effects of alcohol on blood glucose concentration observed in our study and supports our hypothesis for the mechanism behind these effects in Type 1 diabetes. © 2003 Elsevier Science Ireland Ltd. All rights reserved.

Source: Scopus

The effect of alcohol on blood glucose in Type 1 diabetes--metabolic modelling and integration in a decision support system.

Authors: Plougmann, S., Hejlesen, O., Turner, B., Kerr, D. and Cavan, D.

Journal: Int J Med Inform

Volume: 70

Issue: 2-3

Pages: 337-344

ISSN: 1386-5056

DOI: 10.1016/s1386-5056(03)00038-8

Abstract:

INTRODUCTION: We have recently shown, in studies with patients with Type 1 (insulin dependent) diabetes, that alcohol intake at 21:00 h significantly reduced blood glucose values after 10-12 h, compared with control studies with no alcohol. HYPOTHESIS: We hypothesised that this was due to the following effects of alcohol: (1) alcohol metabolism increases NADH, leading to a reduction in hepatic gluconeogenesis; (2) increased glycogen phosphorylase activity depletes hepatic glycogen stores; (3) after the alcohol is metabolised, hepatic insulin sensitivity is increased, leading to the restoration of glycogen stores and reduction in blood glucose levels; and (4) consequently, after several hours, glycogen stores and insulin sensitivity return to normal. RESULTS: A model describing these changes (DiasNet-Alcohol) was implemented into the DiasNet model of human glucose metabolism. Our study suggests that the DiasNet-Alcohol model gives a reasonable approximation of these effects of alcohol on blood glucose concentration observed in our study and supports our hypothesis for the mechanism behind these effects in Type 1 diabetes.

Source: PubMed

The effect of alcohol on blood glucose in Type 1 diabetes - metabolic modelling and integration in a decision support system

Authors: Plougmann, S., Hejlesen, O., Turner, B., Kerr, D. and Cavan, D.

Journal: INTERNATIONAL JOURNAL OF MEDICAL INFORMATICS

Volume: 70

Issue: 2-3

Pages: 337-344

ISSN: 1386-5056

DOI: 10.1016/S1386-5056(03)00038-8

Source: Web of Science (Lite)

The effect of alcohol on blood glucose in Type 1 diabetes—metabolic modelling and integration in a decision support system

Authors: Plougmann, S., Hejlesen, O.K., Turner, B., Kerr, D. and Cavan, D.A.

Journal: International Journal of Medical Informatics

Volume: 70

Pages: 337-344

ISSN: 1386-5056

DOI: 10.1016/S1386-5056(03)00038-8

Abstract:

Introduction: We have recently shown, in studies with patients with Type 1 (insulin dependent) diabetes, that alcohol intake at 21:00 h significantly reduced blood glucose values after 10–12 h, compared with control studies with no alcohol. Hypothesis: We hypothesised that this was due to the following effects of alcohol: (1) alcohol metabolism increases NADH, leading to a reduction in hepatic gluconeogenesis; (2) increased glycogen phosphorylase activity depletes hepatic glycogen stores; (3) after the alcohol is metabolised, hepatic insulin sensitivity is increased, leading to the restoration of glycogen stores and reduction in blood glucose levels; and (4) consequently, after several hours, glycogen stores and insulin sensitivity return to normal. Results: A model describing these changes (DiasNet-Alcohol) was implemented into the DiasNet model of human glucose metabolism. Our study suggests that the DiasNet-Alcohol model gives a reasonable approximation of these effects of alcohol on blood glucose concentration observed in our study and supports our hypothesis for the mechanism behind these effects in Type 1 diabetes.

http://www.sciencedirect.com/science?_ob=ArticleURL&_udi=B6T7S-48NKV3Y-1&_user=1682380&_coverDate=07%2F31%2F2003&_rdoc=28&_fmt=summary&_orig=browse&_srch=doc-info(%23toc%235066%232003%23999299997%23443478%23FLA%23display%23Volume)&_cdi=5066&_sort=d&_docan

Source: Manual

Preferred by: David Kerr

The effect of alcohol on blood glucose in Type 1 diabetes - metabolic modelling and integration in a decision support system.

Authors: Plougmann, S., Hejlesen, O.K., Turner, B., Kerr, D. and Cavan, D.A.

Journal: Int. J. Medical Informatics

Volume: 70

Pages: 337-344

DOI: 10.1016/S1386-5056(03)00038-8

Source: DBLP

The effect of alcohol on blood glucose in Type 1 diabetes--metabolic modelling and integration in a decision support system.

Authors: Plougmann, S., Hejlesen, O., Turner, B., Kerr, D. and Cavan, D.

Journal: International journal of medical informatics

Volume: 70

Issue: 2-3

Pages: 337-344

eISSN: 1872-8243

ISSN: 1386-5056

DOI: 10.1016/s1386-5056(03)00038-8

Abstract:

Introduction

We have recently shown, in studies with patients with Type 1 (insulin dependent) diabetes, that alcohol intake at 21:00 h significantly reduced blood glucose values after 10-12 h, compared with control studies with no alcohol.

Hypothesis

We hypothesised that this was due to the following effects of alcohol: (1) alcohol metabolism increases NADH, leading to a reduction in hepatic gluconeogenesis; (2) increased glycogen phosphorylase activity depletes hepatic glycogen stores; (3) after the alcohol is metabolised, hepatic insulin sensitivity is increased, leading to the restoration of glycogen stores and reduction in blood glucose levels; and (4) consequently, after several hours, glycogen stores and insulin sensitivity return to normal.

Results

A model describing these changes (DiasNet-Alcohol) was implemented into the DiasNet model of human glucose metabolism. Our study suggests that the DiasNet-Alcohol model gives a reasonable approximation of these effects of alcohol on blood glucose concentration observed in our study and supports our hypothesis for the mechanism behind these effects in Type 1 diabetes.

Source: Europe PubMed Central