Joint hypermobility syndrome and symphysis pubis dysfunction

This source preferred by Carol Clark

Authors: Clark, C.J.

Journal: British Journal of Midwifery

Volume: 18

Pages: 92-97

ISSN: 0969-4900

Joint hypermobility syndrome is a multisystem connective tissue disorder presenting as signs and symptoms associated with tissue laxity. Pain enhancement and chronic pain are frequent features. There is anecdotal evidence that some women report the onset of life-long symptoms during their first pregnancy. This review explores the prevalence of symphysis pubis dysfunction in those with joint hypermobility syndrome, mechanical features associated with pelvic instability and the clinical implications.

Midwives play a key role both during and after pregnancy in recognizing and understanding the impact associated with symphysis pubis dysfunction. Further consideration needs to be given to those with joint hypermobility syndrome who are predisposed to soft tissue changes, joint instability and pain.

This data was imported from Scopus:

Authors: Clark, C.

Journal: British Journal of Midwifery

Volume: 18

Issue: 2

Pages: 92-97

ISSN: 0969-4900

DOI: 10.12968/bjom.2010.18.2.46406

Joint hypermobility syndrome is a multisystem connective tissue disorder presenting as signs and symptoms associated with tissue laxity. Pain enhancement and chronic pain are frequent features. There is anecdotal evidence that some women report the onset of life-long symptoms during their first pregnancy. This review explores the prevalence of symphysis pubis dysfunction in those with joint hypermobility syndrome, mechanical features associated with pelvic instability and the clinical implications. Midwives play a key role both during and after pregnancy in recognizing and understanding the impact associated with symphysis pubis dysfunction. Further consideration needs to be given to those with joint hypermobility syndrome who are predisposed to soft tissue changes, joint Instability and pain.

The data on this page was last updated at 05:09 on February 24, 2020.